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        <identifier>oai:fra.repo.nii.ac.jp:02001607</identifier>
        <datestamp>2025-06-30T02:18:33Z</datestamp>
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          <dc:title xml:lang="ja">コーチゾルと成長ホルモンがベニザケの海水適応能に与える影響</dc:title>
          <dc:title xml:lang="en">Effects of Cortisol and Growth Hormone on the Seawater Tolerance of Sockeye Salmon (Oncorhynchus nerka)</dc:title>
          <jpcoar:creator>
            <jpcoar:creatorName xml:lang="ja">伴, 真俊</jpcoar:creatorName>
            <jpcoar:creatorName xml:lang="ja-Kana">バン, マサトシ</jpcoar:creatorName>
            <jpcoar:creatorName xml:lang="en">Ban, Masatoshi</jpcoar:creatorName>
            <jpcoar:familyName xml:lang="ja">伴</jpcoar:familyName>
            <jpcoar:familyName xml:lang="ja-Kana">バン</jpcoar:familyName>
            <jpcoar:familyName xml:lang="en">Ban</jpcoar:familyName>
            <jpcoar:givenName xml:lang="ja">真俊</jpcoar:givenName>
            <jpcoar:givenName xml:lang="ja-Kana">マサトシ</jpcoar:givenName>
            <jpcoar:givenName xml:lang="en">Masatoshi</jpcoar:givenName>
          </jpcoar:creator>
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          <jpcoar:subject xml:lang="en" subjectScheme="Other">sockeye salmon; seawater tolerance; gill Na⁺,K⁺ATPase; cortisol; growth hormone</jpcoar:subject>
          <datacite:description xml:lang="ja" descriptionType="Abstract">コーチゾルと成長ホルモン(GH)が、ベニザケの海水適応能に与える影響を調べるため、体重当たり1μgの濃度に調整した各ホルモンを、腹腔内へ2習慣に亘って注射した。同様の実験を3月と5月の2回行った。3月のコーチゾル群における鰓のNa(+)、K(+)-ATPase活性は、生理食塩水を投与した対照群に比べて約1.5培高い値を示した。海水移行24時間後の血中ナトリウム(Na)濃度は、対照群が191.1mMだったのに対して、コーチゾル群とGH群は各々164.3mMと153.5mMを示した。しかし、5月には3群とも共通して鰓のNa(+)、K(+)-ATPase活性が16.8-17.1μmols Pi/mg pro/hまで上昇するとともに、海水移行後の血中Na濃度は148.0-154.4mMまで低下し、ホルモンの投与効果が認められなかった。これらの結果は、コーチゾルとGHが、ベニザケの海水適応能獲得過程に重要な役割を果たすことを示している。しかし、ホルモンの効果は投与時期により異なることが予想される。</datacite:description>
          <datacite:description xml:lang="en" descriptionType="Abstract">In order to determine hormonal effects on the seawater tolerance of sockeye salmon (Oncorhynchus nerka), cortisol or growth hormone (GH) were administered via intraperitoneal injection at 1 μg per body weight for two weeks in March and May in the laboratory. In March, the gill Na⁺, K⁺-ATPase activity of the cortisol group was 1.5-fold higher than that of the control group injected only with saline. Plasma sodium concentrations 24 h after transfer to seawater for the cortisol and GH groups declined to 164.3 mM and 153.5 mM, respectively, while the control group had 191.1 mM. In May, however, the gill Na⁺, K⁺-ATPase activity in all these treatments elevated to 16.8 - 17.1 μmols Pi/mg pro/h and the plasma sodium concentrations decreased to 148.0 - 154.5 mM. Administration of cortisol and GH had no effect on seawater tolerance in May. These results indicate that cortisol and GH are important hormones promoting seawater tolerance in sockeye salmon, while effects of these hormones may vary with administration time.</datacite:description>
          <dc:publisher xml:lang="ja">さけ・ます資源管理センター</dc:publisher>
          <dc:publisher xml:lang="en">the National Salmon Resources Center</dc:publisher>
          <datacite:date dateType="Issued">2002-12</datacite:date>
          <dc:language>eng</dc:language>
          <dc:type rdf:resource="http://purl.org/coar/resource_type/c_6501">article</dc:type>
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          <jpcoar:identifier identifierType="URI">https://fra.repo.nii.ac.jp/records/2001607</jpcoar:identifier>
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            <jpcoar:relatedIdentifier identifierType="Local">salmon-k-k_5-5</jpcoar:relatedIdentifier>
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          <jpcoar:sourceIdentifier identifierType="NCID">AA11339120</jpcoar:sourceIdentifier>
          <jpcoar:sourceIdentifier identifierType="PISSN">1344-7556</jpcoar:sourceIdentifier>
          <jpcoar:sourceTitle xml:lang="ja">さけ・ます資源管理センター研究報告</jpcoar:sourceTitle>
          <jpcoar:sourceTitle xml:lang="en">Bulletin of the National Salmon Resources Center</jpcoar:sourceTitle>
          <jpcoar:volume>5</jpcoar:volume>
          <jpcoar:numPages>5</jpcoar:numPages>
          <jpcoar:pageStart>27</jpcoar:pageStart>
          <jpcoar:pageEnd>31</jpcoar:pageEnd>
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            <datacite:date dateType="Available">2024-04-05</datacite:date>
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